Epilepsy and Seizure News
Neurofeedback News
Rewiring Your Brain: Neurofeedback Goes Mainstream -
See more of our trusted coverage when you search. Prefer Newsweek on Google to see more of our trusted coverage when you search. Will Strahl walked up to my door with a massive black briefcase in his ...
What Is Neurofeedback Therapy And How Does It Work? -
Ashwini Nadkarni, M.D., is a board-certified psychiatrist, Assistant Professor at Harvard Medical School and Interim Vice Chair for Faculty Affairs in the Department of Psychiatry at Brigham and Women ...
What Is Neurofeedback? -
Whenever I mention that neurofeedback helped me regain my life after my multiple brain injuries — or that I’m now a neurofeedback provider — the first reaction is to ask me, what is neurofeedback? I’m ...
What Does a Neurofeedback Technician Do? -
Neurofeedback is about helping a client's brain learn to self-regulate by showing it its own EEG activity on a screen or through headphones. It is like going to the gym for your brain. It is not a ...
Neurofeedback for ADHD: Significant, Lasting Improvement -
Computer-based neurofeedback can produce significant and lasting improvement in attention and focus in children with attention-deficit/hyperactivity disorder (ADHD ...
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American Epilepsy Society/Congress of Neurological Surgeons Epilepsy Surgery Clinical Practice Guideline -
This practice guideline from the American Epilepsy Society and the Congress of Neurological Surgeons provides evidence-based recommendations for surgical treatment of epilepsy for adults and children. It is an update to previous practice parameters published by the Academy of Neurology in 2003. The multidisciplinary panel addressed resective procedures for mesial temporal epilepsy, neocortical ...
Dual-channel fluorescence imaging revealing epilepsy-mediated ferroptosis processes in the mouse brain -
Epileptic seizures are caused by abnormal electrical activity in the brain. Although many underlying disease mechanisms may lead to epilepsy, the cause of the disease remains unknown in about 50% of patients worldwide. Given the complex interactions involving multiple signaling molecules in the course of epilepsy, relying solely on a single molecule for detection may lead to false positive resu...
Systems Toxicology Indicates AKT1/PTGS2-Centered Neuroinflammatory-Mitochondrial Crosstalk in Lidocaine Associated Neurotoxicity -
CONCLUSIONS: Lidocaine-associated neurotoxicity may involve an AKT1/PTGS2-centered neuroinflammatory-mitochondrial crosstalk network accompanied by synaptic dysfunction. This study provides a systems-level mechanistic insight into lidocaine neurotoxicity and identifies potential targets for improving the safety of pediatric lidocaine-containing formulations.
Creb1 aggravates glutamate-induced excitotoxicity and KA-induced seizure activity by transcriptionally activating Timp1 -
Epilepsy is a chronic neurological disorder, more common in children and the elderly. Although treatments have advanced, significant challenges remain. This study focuses on its molecular mechanisms to provide new insights and strategies for its treatment. Gene expression omnibus (GEO) database analysis, enrichment analysis, and the establishment of protein-protein interaction (PPI) networks we...
Assessment of fine motor performance and motor speed in pediatric patients with juvenile myoclonic epilepsy -
CONCLUSIONS: Adolescents with JME showed impaired fine motor performance, particularly in patients with uncontrolled seizures and combination ASMs therapy. The 9-HPT may be a useful simple tool for assessing motor dysfunction in JME.
The dual role of BDNF in epilepsy: insights from calpain-mediated TrkB-FL cleavage -
Despite the efficacy of current antiseizure medications in managing seizures, approximately one-third of patients develop refractory epilepsy, highlighting an urgent need to uncover the molecular mechanisms driving epileptogenesis. This process emerges from a complex interplay of molecular alterations, involving changes in neuronal signaling pathways. Among these, brain-derived neurotrophic fac...
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